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Comparison of antiemetic efficacy and safety of palonosetron vs ondansetron in the prevention of chemotherapy-induced nausea and vomiting in children

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Comparison of antiemetic efficacy and safety of palonosetron vs ondansetron in the prevention of chemotherapy-induced nausea and vomiting in children

Background Chemotherapy-induced nausea and vomiting (CINV) in children is a major side effect despite the use of combination antiemetic drugs.

Objective To compare the efficacy and safety profile of palonosetron, a second-generation 5-hydroxytryptamine-3 (5-HT3) receptor antagonist, with ondansetron in the prevention of CINV in children.

Methods A prospective, randomized, crossover study was conducted in patients aged 2-18 years. 160 chemotherapy cycles, consisting of chemotherapy drugs with moderate- and high-emetogenic potential, were studied. The study group received a single dose of intravenous (IV) palonosetron 5 mcg/kg, and the standard group received IV ondansetron 5 mg/m2 every 8 hours while receiving chemotherapy. The patients were observed for vomiting, use of rescue antiemetic medications, and nausea from Day 1 0-72 hours after completion of each chemotherapy cycle. All adverse events during the study period were recorded.

Results The overall percentage of patients with complete response (CR) in the palonosetron and ondansetron groups were 60% and 56.2%, respectively (P = .631). The CR rates in the palonosetron and ondansetron groups were 75% and 70%, respectively, in the acute phase (P = .479), and 68.8% and 65%, respectively, in the delayed phase (P = .614). There was no statistically significant difference in the CR rates cross both groups.

Conclusion A single dose of palonosetron is noninferior to ondansetron in the prevention of CINV in children and can be considered as an alternative antiemetic drug. There was no significant difference in adverse effects between the palonosetron and ondansetron group. 

 

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The Journal of Community and Supportive Oncology - 13(6)
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209-213
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chemotherapy-induced nausea and vomiting, CINV, antiemetic drugs, palonosetron, 5-hydroxytryptamine-3, 5-HT3, ondansetron
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Background Chemotherapy-induced nausea and vomiting (CINV) in children is a major side effect despite the use of combination antiemetic drugs.

Objective To compare the efficacy and safety profile of palonosetron, a second-generation 5-hydroxytryptamine-3 (5-HT3) receptor antagonist, with ondansetron in the prevention of CINV in children.

Methods A prospective, randomized, crossover study was conducted in patients aged 2-18 years. 160 chemotherapy cycles, consisting of chemotherapy drugs with moderate- and high-emetogenic potential, were studied. The study group received a single dose of intravenous (IV) palonosetron 5 mcg/kg, and the standard group received IV ondansetron 5 mg/m2 every 8 hours while receiving chemotherapy. The patients were observed for vomiting, use of rescue antiemetic medications, and nausea from Day 1 0-72 hours after completion of each chemotherapy cycle. All adverse events during the study period were recorded.

Results The overall percentage of patients with complete response (CR) in the palonosetron and ondansetron groups were 60% and 56.2%, respectively (P = .631). The CR rates in the palonosetron and ondansetron groups were 75% and 70%, respectively, in the acute phase (P = .479), and 68.8% and 65%, respectively, in the delayed phase (P = .614). There was no statistically significant difference in the CR rates cross both groups.

Conclusion A single dose of palonosetron is noninferior to ondansetron in the prevention of CINV in children and can be considered as an alternative antiemetic drug. There was no significant difference in adverse effects between the palonosetron and ondansetron group. 

 

Click on the PDF icon at the top of this introduction to read the full article.

Background Chemotherapy-induced nausea and vomiting (CINV) in children is a major side effect despite the use of combination antiemetic drugs.

Objective To compare the efficacy and safety profile of palonosetron, a second-generation 5-hydroxytryptamine-3 (5-HT3) receptor antagonist, with ondansetron in the prevention of CINV in children.

Methods A prospective, randomized, crossover study was conducted in patients aged 2-18 years. 160 chemotherapy cycles, consisting of chemotherapy drugs with moderate- and high-emetogenic potential, were studied. The study group received a single dose of intravenous (IV) palonosetron 5 mcg/kg, and the standard group received IV ondansetron 5 mg/m2 every 8 hours while receiving chemotherapy. The patients were observed for vomiting, use of rescue antiemetic medications, and nausea from Day 1 0-72 hours after completion of each chemotherapy cycle. All adverse events during the study period were recorded.

Results The overall percentage of patients with complete response (CR) in the palonosetron and ondansetron groups were 60% and 56.2%, respectively (P = .631). The CR rates in the palonosetron and ondansetron groups were 75% and 70%, respectively, in the acute phase (P = .479), and 68.8% and 65%, respectively, in the delayed phase (P = .614). There was no statistically significant difference in the CR rates cross both groups.

Conclusion A single dose of palonosetron is noninferior to ondansetron in the prevention of CINV in children and can be considered as an alternative antiemetic drug. There was no significant difference in adverse effects between the palonosetron and ondansetron group. 

 

Click on the PDF icon at the top of this introduction to read the full article.

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The Journal of Community and Supportive Oncology - 13(6)
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The Journal of Community and Supportive Oncology - 13(6)
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209-213
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Comparison of antiemetic efficacy and safety of palonosetron vs ondansetron in the prevention of chemotherapy-induced nausea and vomiting in children
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Comparison of antiemetic efficacy and safety of palonosetron vs ondansetron in the prevention of chemotherapy-induced nausea and vomiting in children
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chemotherapy-induced nausea and vomiting, CINV, antiemetic drugs, palonosetron, 5-hydroxytryptamine-3, 5-HT3, ondansetron
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Olaparib for BRCA-mutated advanced ovarian cancer

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Olaparib for BRCA-mutated advanced ovarian cancer
Epithelial ovarian cancer is the leading cause of death from gynecologic cancer in the United States and the fifth most common cause of cancer-related mortality. Treatment options, particularly for tumors with mutations in the breast cancer susceptibility genes, BRCA1 and BRCA2, are currently limited, and significant research has focused on the development of novel therapies. Inhibitors of poly(ADP)ribose polymerase (PARP), which specifically target BRCA-mutant cancer cells by exploiting the defective DNA repair pathways inherent in these tumors, have proven particularly promising, though clinical development has not been without challenges. Development of olaparib was halted in 2011 following disappointing clinical trial results, but the manufacturer resurrected the drug following retrospective analyses in patients with BRCA1/2 mutations. 

 

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Epithelial ovarian cancer is the leading cause of death from gynecologic cancer in the United States and the fifth most common cause of cancer-related mortality. Treatment options, particularly for tumors with mutations in the breast cancer susceptibility genes, BRCA1 and BRCA2, are currently limited, and significant research has focused on the development of novel therapies. Inhibitors of poly(ADP)ribose polymerase (PARP), which specifically target BRCA-mutant cancer cells by exploiting the defective DNA repair pathways inherent in these tumors, have proven particularly promising, though clinical development has not been without challenges. Development of olaparib was halted in 2011 following disappointing clinical trial results, but the manufacturer resurrected the drug following retrospective analyses in patients with BRCA1/2 mutations. 

 

Click on the PDF icon at the top of this introduction to read the full article.

 
Epithelial ovarian cancer is the leading cause of death from gynecologic cancer in the United States and the fifth most common cause of cancer-related mortality. Treatment options, particularly for tumors with mutations in the breast cancer susceptibility genes, BRCA1 and BRCA2, are currently limited, and significant research has focused on the development of novel therapies. Inhibitors of poly(ADP)ribose polymerase (PARP), which specifically target BRCA-mutant cancer cells by exploiting the defective DNA repair pathways inherent in these tumors, have proven particularly promising, though clinical development has not been without challenges. Development of olaparib was halted in 2011 following disappointing clinical trial results, but the manufacturer resurrected the drug following retrospective analyses in patients with BRCA1/2 mutations. 

 

Click on the PDF icon at the top of this introduction to read the full article.

 
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The Journal of Community and Supportive Oncology - 13(6)
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The Journal of Community and Supportive Oncology - 13(6)
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Olaparib for BRCA-mutated advanced ovarian cancer
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It is not your mother/father’s ASCO anymore…

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It is not your mother/father’s ASCO anymore…
Well, what just happened in Chicago? Nearly 30,000 of our closest friends and colleagues descended on the windy city to meet in the McCormick Center to catch up on the latest data and updates in oncology at the American Society of Clinical Oncology's annual conference. What a marathon meeting it was. Who remembers when the meeting was held in one hotel (can you believe it), then two hotels, and then eventually shifted to convention centers to accommodate the burgeoning numbers of attendees? The program format is also quite different now than it was at the outset.
 
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Well, what just happened in Chicago? Nearly 30,000 of our closest friends and colleagues descended on the windy city to meet in the McCormick Center to catch up on the latest data and updates in oncology at the American Society of Clinical Oncology's annual conference. What a marathon meeting it was. Who remembers when the meeting was held in one hotel (can you believe it), then two hotels, and then eventually shifted to convention centers to accommodate the burgeoning numbers of attendees? The program format is also quite different now than it was at the outset.
 
Click on the PDF icon at the top of this introduction to read the full article.
 
Well, what just happened in Chicago? Nearly 30,000 of our closest friends and colleagues descended on the windy city to meet in the McCormick Center to catch up on the latest data and updates in oncology at the American Society of Clinical Oncology's annual conference. What a marathon meeting it was. Who remembers when the meeting was held in one hotel (can you believe it), then two hotels, and then eventually shifted to convention centers to accommodate the burgeoning numbers of attendees? The program format is also quite different now than it was at the outset.
 
Click on the PDF icon at the top of this introduction to read the full article.
 
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The Journal of Community and Supportive Oncology - 13(6)
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The Journal of Community and Supportive Oncology - 13(6)
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It is not your mother/father’s ASCO anymore…
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David Henry's JCSO podcast, May 2015

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David Henry's JCSO podcast, May 2015

In his monthly podcast for The Journal of Community and Supportive Oncology, Dr David Henry looks at Original Reports on the treatment of metastatic breast cancer with nab-paclitaxel in the community practice setting; health professionals’ attitudes toward detecting and managing cancer-related anorexia-cachexia syndrome; the factors associated with symptom-related emergency department visits and hospital admissions during ambulatory cancer treatment; and differences in treatment between urban and rural women with hormone receptor-positive early-stage breast cancer. He also discusses a case report on a patient with severe chemotherapy-induced peripheral neuropathy who experienced a significant response to lacosamide, and Community Translations item using blinatumomab for hard-to-treat acute lymphoblastic leukemia.  

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In his monthly podcast for The Journal of Community and Supportive Oncology, Dr David Henry looks at Original Reports on the treatment of metastatic breast cancer with nab-paclitaxel in the community practice setting; health professionals’ attitudes toward detecting and managing cancer-related anorexia-cachexia syndrome; the factors associated with symptom-related emergency department visits and hospital admissions during ambulatory cancer treatment; and differences in treatment between urban and rural women with hormone receptor-positive early-stage breast cancer. He also discusses a case report on a patient with severe chemotherapy-induced peripheral neuropathy who experienced a significant response to lacosamide, and Community Translations item using blinatumomab for hard-to-treat acute lymphoblastic leukemia.  

In his monthly podcast for The Journal of Community and Supportive Oncology, Dr David Henry looks at Original Reports on the treatment of metastatic breast cancer with nab-paclitaxel in the community practice setting; health professionals’ attitudes toward detecting and managing cancer-related anorexia-cachexia syndrome; the factors associated with symptom-related emergency department visits and hospital admissions during ambulatory cancer treatment; and differences in treatment between urban and rural women with hormone receptor-positive early-stage breast cancer. He also discusses a case report on a patient with severe chemotherapy-induced peripheral neuropathy who experienced a significant response to lacosamide, and Community Translations item using blinatumomab for hard-to-treat acute lymphoblastic leukemia.  

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David Henry's JCSO podcast, May 2015
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David Henry's JCSO podcast, May 2015
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blinatumomab, relapsed/ refractory, Philadelphia chromosome-negative, B-cell acute lymphoblastic leukemia,
BCP-ALL, CD19, CD3, dosage-schedule, nab-paclitaxel, metastatic breast cancer, MBC, human epidermal growth factor receptor 2, HER2–negative, anorexia-cachexia syndrome, CACS, non-small-cell lung cancer, NSCLC, symptom distress, quality of life, QoL, side effects, emergency department, ED, hospital admissions, HAs, emergency department visits, EDVs, symptom-related EDVs, sx-EDV/HAs, urban, rural, hormone receptor-positive, breast cancer, lacosamide, anticonvulsant, chemotherapy-induced peripheral neuropathy, CIPN, urothelial carcinoma,
MVAC, methotrexate, vincristine, adriamycin, cisplatin
Legacy Keywords
blinatumomab, relapsed/ refractory, Philadelphia chromosome-negative, B-cell acute lymphoblastic leukemia,
BCP-ALL, CD19, CD3, dosage-schedule, nab-paclitaxel, metastatic breast cancer, MBC, human epidermal growth factor receptor 2, HER2–negative, anorexia-cachexia syndrome, CACS, non-small-cell lung cancer, NSCLC, symptom distress, quality of life, QoL, side effects, emergency department, ED, hospital admissions, HAs, emergency department visits, EDVs, symptom-related EDVs, sx-EDV/HAs, urban, rural, hormone receptor-positive, breast cancer, lacosamide, anticonvulsant, chemotherapy-induced peripheral neuropathy, CIPN, urothelial carcinoma,
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Significant response to lacosamide in a patient with severe chemotherapy-induced peripheral neuropathy

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Significant response to lacosamide in a patient with severe chemotherapy-induced peripheral neuropathy
Chemotherapy-induced peripheral neuropathy (CIPN) is a major dose-limiting toxicity of potentially curative cancer therapy regimens. Cisplatin is the class of chemotherapy agent that has a broad spectrum of activity against several solid tumors, but it induces sensory neuropathy of upper and lower extremities. Cisplatin-induced peripheral neuropathy is usually in a “gloves and socks” distribution that can persist for months or years after completion of chemotherapy treatment. If the pain is severe, it affects the patient’s long-term quality of life and can potentially result in chemotherapy dose reduction or treatment discontinuation.
 

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The Journal of Community and Supportive Oncology - 13(5)
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202-204
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peripheral neuropathy, chemotherapy-induced peripheral neuropathy, CIPN, lacosamide, cisplatin, MVAC, methotrexate, vincristine, adriamycin, and cisplatin,
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Chemotherapy-induced peripheral neuropathy (CIPN) is a major dose-limiting toxicity of potentially curative cancer therapy regimens. Cisplatin is the class of chemotherapy agent that has a broad spectrum of activity against several solid tumors, but it induces sensory neuropathy of upper and lower extremities. Cisplatin-induced peripheral neuropathy is usually in a “gloves and socks” distribution that can persist for months or years after completion of chemotherapy treatment. If the pain is severe, it affects the patient’s long-term quality of life and can potentially result in chemotherapy dose reduction or treatment discontinuation.
 

Click on the PDF icon at the top of this introduction to read the full article.

 

Chemotherapy-induced peripheral neuropathy (CIPN) is a major dose-limiting toxicity of potentially curative cancer therapy regimens. Cisplatin is the class of chemotherapy agent that has a broad spectrum of activity against several solid tumors, but it induces sensory neuropathy of upper and lower extremities. Cisplatin-induced peripheral neuropathy is usually in a “gloves and socks” distribution that can persist for months or years after completion of chemotherapy treatment. If the pain is severe, it affects the patient’s long-term quality of life and can potentially result in chemotherapy dose reduction or treatment discontinuation.
 

Click on the PDF icon at the top of this introduction to read the full article.

 

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The Journal of Community and Supportive Oncology - 13(5)
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The Journal of Community and Supportive Oncology - 13(5)
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202-204
Page Number
202-204
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Significant response to lacosamide in a patient with severe chemotherapy-induced peripheral neuropathy
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Significant response to lacosamide in a patient with severe chemotherapy-induced peripheral neuropathy
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peripheral neuropathy, chemotherapy-induced peripheral neuropathy, CIPN, lacosamide, cisplatin, MVAC, methotrexate, vincristine, adriamycin, and cisplatin,
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peripheral neuropathy, chemotherapy-induced peripheral neuropathy, CIPN, lacosamide, cisplatin, MVAC, methotrexate, vincristine, adriamycin, and cisplatin,
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Treatment differences between urban and rural women with hormone receptor-positive early-stage breast cancer based on 21-gene assay recurrence score result

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Treatment differences between urban and rural women with hormone receptor-positive early-stage breast cancer based on 21-gene assay recurrence score result
Background Women who live in rural and urban settings have different outcomes for breast cancer. A 21-gene assay predicts 10- year distant recurrence risk and potential benefit of chemotherapy for women with hormone receptor-positive (HR+) breast cancer.

 

Objective To assess differences in scores and cancer therapies received by rural versus urban residence. 

 

Methods We conducted a multi-institutional retrospective chart review of breast cancer patients diagnosed 2005-2010 with score results. Comparisons by rural versus urban residence (determined by rural-urban commuting area (RUCA) codes derived from zip codes) were made using the Fisher exact test for discrete data such as recurrence score results (<18 vs >18; score range, 0-100, with lower results correlated with less risk of distant recurrence), stage, and receptor status. The Wilcoxon rank sum test was used for continuous data (score results 0-100 and age.) All tests were at a 2-sided significance level of .05.

 

Results 504 patients had RUCA codes (92% white, 62% postmenopausal). For rural (n = 135) compared with urban (n = 369) patients, the median scores were 16 and 18, respectively, P = .18. Most of the patients received endocrine therapy, 123 of 135 (91%) rural, compared with 339 of 369 (92%) urban (P = .19). For scores 18-30, 20 of 56 (36%) rural patients, compared with 82 of 159 (52%) urban patients received chemotherapy (P = .03).

 

Limitations Limitations include lack of randomization to receipt of the assay.

 

Conclusions Recurrence score results did not significantly differ between women based on residence, although women living in a rural area received significantly less chemotherapy for scores >18. This suggests that for HR-positive breast cancer, discrepancies between rural and urban residence are driven by treatment factors rather than differences in biology.

 

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The Journal of Community and Supportive Oncology - 13(5)
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hormone receptor-positive, HR, breast cancer, rural patients, urban patients, recurrence rates, chemotherapy
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Background Women who live in rural and urban settings have different outcomes for breast cancer. A 21-gene assay predicts 10- year distant recurrence risk and potential benefit of chemotherapy for women with hormone receptor-positive (HR+) breast cancer.

 

Objective To assess differences in scores and cancer therapies received by rural versus urban residence. 

 

Methods We conducted a multi-institutional retrospective chart review of breast cancer patients diagnosed 2005-2010 with score results. Comparisons by rural versus urban residence (determined by rural-urban commuting area (RUCA) codes derived from zip codes) were made using the Fisher exact test for discrete data such as recurrence score results (<18 vs >18; score range, 0-100, with lower results correlated with less risk of distant recurrence), stage, and receptor status. The Wilcoxon rank sum test was used for continuous data (score results 0-100 and age.) All tests were at a 2-sided significance level of .05.

 

Results 504 patients had RUCA codes (92% white, 62% postmenopausal). For rural (n = 135) compared with urban (n = 369) patients, the median scores were 16 and 18, respectively, P = .18. Most of the patients received endocrine therapy, 123 of 135 (91%) rural, compared with 339 of 369 (92%) urban (P = .19). For scores 18-30, 20 of 56 (36%) rural patients, compared with 82 of 159 (52%) urban patients received chemotherapy (P = .03).

 

Limitations Limitations include lack of randomization to receipt of the assay.

 

Conclusions Recurrence score results did not significantly differ between women based on residence, although women living in a rural area received significantly less chemotherapy for scores >18. This suggests that for HR-positive breast cancer, discrepancies between rural and urban residence are driven by treatment factors rather than differences in biology.

 

Click on the PDF icon at the top of this introduction to read the full article.

 

Background Women who live in rural and urban settings have different outcomes for breast cancer. A 21-gene assay predicts 10- year distant recurrence risk and potential benefit of chemotherapy for women with hormone receptor-positive (HR+) breast cancer.

 

Objective To assess differences in scores and cancer therapies received by rural versus urban residence. 

 

Methods We conducted a multi-institutional retrospective chart review of breast cancer patients diagnosed 2005-2010 with score results. Comparisons by rural versus urban residence (determined by rural-urban commuting area (RUCA) codes derived from zip codes) were made using the Fisher exact test for discrete data such as recurrence score results (<18 vs >18; score range, 0-100, with lower results correlated with less risk of distant recurrence), stage, and receptor status. The Wilcoxon rank sum test was used for continuous data (score results 0-100 and age.) All tests were at a 2-sided significance level of .05.

 

Results 504 patients had RUCA codes (92% white, 62% postmenopausal). For rural (n = 135) compared with urban (n = 369) patients, the median scores were 16 and 18, respectively, P = .18. Most of the patients received endocrine therapy, 123 of 135 (91%) rural, compared with 339 of 369 (92%) urban (P = .19). For scores 18-30, 20 of 56 (36%) rural patients, compared with 82 of 159 (52%) urban patients received chemotherapy (P = .03).

 

Limitations Limitations include lack of randomization to receipt of the assay.

 

Conclusions Recurrence score results did not significantly differ between women based on residence, although women living in a rural area received significantly less chemotherapy for scores >18. This suggests that for HR-positive breast cancer, discrepancies between rural and urban residence are driven by treatment factors rather than differences in biology.

 

Click on the PDF icon at the top of this introduction to read the full article.

 

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The Journal of Community and Supportive Oncology - 13(5)
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The Journal of Community and Supportive Oncology - 13(5)
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195-201
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195-201
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Treatment differences between urban and rural women with hormone receptor-positive early-stage breast cancer based on 21-gene assay recurrence score result
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Treatment differences between urban and rural women with hormone receptor-positive early-stage breast cancer based on 21-gene assay recurrence score result
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Symptom-related emergency department visits and hospital admissions during ambulatory cancer treatment

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Symptom-related emergency department visits and hospital admissions during ambulatory cancer treatment

Background People with cancer experience symptoms related to the disease and treatments. Symptom distress has a negative impact on quality of life (QoL). Attending to symptoms and side effects of treatment promotes safe and effective delivery of therapies and may prevent or reduce emergency department visits (EDVs) and unplanned hospital admissions (HAs). There is limited evidence examining symptom-related EDVs or HAs (sx-EDV/HAs) and interventions in ambulatory oncology patients.

Objective To examine factors associated with sx-EDV/HAs in ambulatory oncology patients receiving chemotherapy and/or radiation.

Methods This secondary analysis used data from a randomized controlled trial of ambulatory oncology patients (n = 663) who received the web-based Electronic Self-Report Assessment – Cancer intervention (symptom self-monitoring, tailored education, and communication coaching) or usual care with symptom self-monitoring alone. Group differences were described by summary statistics and compared by t test. Factors associated with the odds of at least 1 sx-EDV/HA were modeled using logistic regression.

Results 98 patients had a total of 171 sx-EDV/HAs with no difference between groups. Higher odds of at least 1 sx-EDV/HA were associated with socioeconomic and clinical factors. The multivariable model indicated that work status, education level, treatment modality, and on-treatment Symptom Distress Scale-15 scores were significantly associated with having at least 1 sx-EDV/HA.

Limitations This is a secondary analysis not sized to determine cause and effect. The results have limited generalizability.

Conclusion Most patients did not experience a sx-EDV/HA. Demographic and clinical factors predicted a sx-EDV/HA.

Funding National Institute of Nursing Research, National Institutes of Health, R01 NR008726; 2008-2011

 

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Background People with cancer experience symptoms related to the disease and treatments. Symptom distress has a negative impact on quality of life (QoL). Attending to symptoms and side effects of treatment promotes safe and effective delivery of therapies and may prevent or reduce emergency department visits (EDVs) and unplanned hospital admissions (HAs). There is limited evidence examining symptom-related EDVs or HAs (sx-EDV/HAs) and interventions in ambulatory oncology patients.

Objective To examine factors associated with sx-EDV/HAs in ambulatory oncology patients receiving chemotherapy and/or radiation.

Methods This secondary analysis used data from a randomized controlled trial of ambulatory oncology patients (n = 663) who received the web-based Electronic Self-Report Assessment – Cancer intervention (symptom self-monitoring, tailored education, and communication coaching) or usual care with symptom self-monitoring alone. Group differences were described by summary statistics and compared by t test. Factors associated with the odds of at least 1 sx-EDV/HA were modeled using logistic regression.

Results 98 patients had a total of 171 sx-EDV/HAs with no difference between groups. Higher odds of at least 1 sx-EDV/HA were associated with socioeconomic and clinical factors. The multivariable model indicated that work status, education level, treatment modality, and on-treatment Symptom Distress Scale-15 scores were significantly associated with having at least 1 sx-EDV/HA.

Limitations This is a secondary analysis not sized to determine cause and effect. The results have limited generalizability.

Conclusion Most patients did not experience a sx-EDV/HA. Demographic and clinical factors predicted a sx-EDV/HA.

Funding National Institute of Nursing Research, National Institutes of Health, R01 NR008726; 2008-2011

 

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Background People with cancer experience symptoms related to the disease and treatments. Symptom distress has a negative impact on quality of life (QoL). Attending to symptoms and side effects of treatment promotes safe and effective delivery of therapies and may prevent or reduce emergency department visits (EDVs) and unplanned hospital admissions (HAs). There is limited evidence examining symptom-related EDVs or HAs (sx-EDV/HAs) and interventions in ambulatory oncology patients.

Objective To examine factors associated with sx-EDV/HAs in ambulatory oncology patients receiving chemotherapy and/or radiation.

Methods This secondary analysis used data from a randomized controlled trial of ambulatory oncology patients (n = 663) who received the web-based Electronic Self-Report Assessment – Cancer intervention (symptom self-monitoring, tailored education, and communication coaching) or usual care with symptom self-monitoring alone. Group differences were described by summary statistics and compared by t test. Factors associated with the odds of at least 1 sx-EDV/HA were modeled using logistic regression.

Results 98 patients had a total of 171 sx-EDV/HAs with no difference between groups. Higher odds of at least 1 sx-EDV/HA were associated with socioeconomic and clinical factors. The multivariable model indicated that work status, education level, treatment modality, and on-treatment Symptom Distress Scale-15 scores were significantly associated with having at least 1 sx-EDV/HA.

Limitations This is a secondary analysis not sized to determine cause and effect. The results have limited generalizability.

Conclusion Most patients did not experience a sx-EDV/HA. Demographic and clinical factors predicted a sx-EDV/HA.

Funding National Institute of Nursing Research, National Institutes of Health, R01 NR008726; 2008-2011

 

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The Journal of Community and Supportive Oncology - 13(5)
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The Journal of Community and Supportive Oncology - 13(5)
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188-194
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188-194
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Symptom-related emergency department visits and hospital admissions during ambulatory cancer treatment
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symptom distress, side effects, quality of life, QoL, emergency department visits, EDVs, hospital admissions
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Health professionals’ attitudes toward the detection and management of cancer-related anorexia-cachexia syndrome, and a proposal for standardized assessment

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Health professionals’ attitudes toward the detection and management of cancer-related anorexia-cachexia syndrome, and a proposal for standardized assessment
Background The identification and management of patients with cancer anorexia-cachexia syndrome (CACS) can be a challenge despite recent international consensus on the definition of the condition.

 
Objectives To describe the current views and practice patterns of community oncologists and oncology nurses in regard to CACS and to propose a standardized, pragmatic assessment of CACS for oncological practice.

 
Methods and materials Responses from 151 community oncologists and nurses obtained across 5 surveys were analyzed. Questions addressed CACS in general and in patients with non-small-cell lung cancer (NSCLC). Surveys 1-3 were directed at physicians, and surveys 4 and 5 were directed at nurses. Surveys 1, 2, 4, and 5 focused on the recognition and monitoring of CACS, and Survey 3 on symptom management.

 
Results 67% of medical oncologists in Survey 3 selected weight loss as the most important criterion for diagnosing CACS and cited declining appetite and performance status (PS) as the most bothersome effects for patients and families. Weight maintenance/gain was the primary treatment objective for oncologists. Respondents to surveys 1 and 2 acknowledged the risk for CACS is high (60%) in NSCLC but considered the risk much lower (4%) in patients completing a first course of therapy with good PS. 91% of oncologists in Survey 3 reported that symptoms that had an impact on calorie intake were important/very important, and 73% were willing to consider a symptom assessment instrument that included appetite. Nurses in surveys 4 and 5 reported weight loss and appetite were most commonly used to identify cachexia. They considered responsibility for the initial assessment of cachexia was the oncologist’s (32%), followed by the nurse practitioner (28%), and the nurse (16%).


Conclusion Most oncologists and nurses recognize the core criteria for the CACS, although there may be under-recognition of the condition’s prevalence, particularly earlier in the course of treatment. There is considerable interest in adopting a brief assessment tool for screening, management, and referral of patient who are affected by or at-risk of CACS.

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The Journal of Community and Supportive Oncology - 13(5)
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181-187
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cancer-related anorexia-cachexia syndrome, CACS, non-small-cell lung cancer, NSCLC, symptom management
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Background The identification and management of patients with cancer anorexia-cachexia syndrome (CACS) can be a challenge despite recent international consensus on the definition of the condition.

 
Objectives To describe the current views and practice patterns of community oncologists and oncology nurses in regard to CACS and to propose a standardized, pragmatic assessment of CACS for oncological practice.

 
Methods and materials Responses from 151 community oncologists and nurses obtained across 5 surveys were analyzed. Questions addressed CACS in general and in patients with non-small-cell lung cancer (NSCLC). Surveys 1-3 were directed at physicians, and surveys 4 and 5 were directed at nurses. Surveys 1, 2, 4, and 5 focused on the recognition and monitoring of CACS, and Survey 3 on symptom management.

 
Results 67% of medical oncologists in Survey 3 selected weight loss as the most important criterion for diagnosing CACS and cited declining appetite and performance status (PS) as the most bothersome effects for patients and families. Weight maintenance/gain was the primary treatment objective for oncologists. Respondents to surveys 1 and 2 acknowledged the risk for CACS is high (60%) in NSCLC but considered the risk much lower (4%) in patients completing a first course of therapy with good PS. 91% of oncologists in Survey 3 reported that symptoms that had an impact on calorie intake were important/very important, and 73% were willing to consider a symptom assessment instrument that included appetite. Nurses in surveys 4 and 5 reported weight loss and appetite were most commonly used to identify cachexia. They considered responsibility for the initial assessment of cachexia was the oncologist’s (32%), followed by the nurse practitioner (28%), and the nurse (16%).


Conclusion Most oncologists and nurses recognize the core criteria for the CACS, although there may be under-recognition of the condition’s prevalence, particularly earlier in the course of treatment. There is considerable interest in adopting a brief assessment tool for screening, management, and referral of patient who are affected by or at-risk of CACS.

Supplemental material


Click on the PDF icon at the top of this introduction to read the full article.
Background The identification and management of patients with cancer anorexia-cachexia syndrome (CACS) can be a challenge despite recent international consensus on the definition of the condition.

 
Objectives To describe the current views and practice patterns of community oncologists and oncology nurses in regard to CACS and to propose a standardized, pragmatic assessment of CACS for oncological practice.

 
Methods and materials Responses from 151 community oncologists and nurses obtained across 5 surveys were analyzed. Questions addressed CACS in general and in patients with non-small-cell lung cancer (NSCLC). Surveys 1-3 were directed at physicians, and surveys 4 and 5 were directed at nurses. Surveys 1, 2, 4, and 5 focused on the recognition and monitoring of CACS, and Survey 3 on symptom management.

 
Results 67% of medical oncologists in Survey 3 selected weight loss as the most important criterion for diagnosing CACS and cited declining appetite and performance status (PS) as the most bothersome effects for patients and families. Weight maintenance/gain was the primary treatment objective for oncologists. Respondents to surveys 1 and 2 acknowledged the risk for CACS is high (60%) in NSCLC but considered the risk much lower (4%) in patients completing a first course of therapy with good PS. 91% of oncologists in Survey 3 reported that symptoms that had an impact on calorie intake were important/very important, and 73% were willing to consider a symptom assessment instrument that included appetite. Nurses in surveys 4 and 5 reported weight loss and appetite were most commonly used to identify cachexia. They considered responsibility for the initial assessment of cachexia was the oncologist’s (32%), followed by the nurse practitioner (28%), and the nurse (16%).


Conclusion Most oncologists and nurses recognize the core criteria for the CACS, although there may be under-recognition of the condition’s prevalence, particularly earlier in the course of treatment. There is considerable interest in adopting a brief assessment tool for screening, management, and referral of patient who are affected by or at-risk of CACS.

Supplemental material


Click on the PDF icon at the top of this introduction to read the full article.
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The Journal of Community and Supportive Oncology - 13(5)
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181-187
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Health professionals’ attitudes toward the detection and management of cancer-related anorexia-cachexia syndrome, and a proposal for standardized assessment
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Health professionals’ attitudes toward the detection and management of cancer-related anorexia-cachexia syndrome, and a proposal for standardized assessment
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cancer-related anorexia-cachexia syndrome, CACS, non-small-cell lung cancer, NSCLC, symptom management
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Treatment of metastatic breast cancer with nab-paclitaxel in the community practice setting: a US oncology survey

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Treatment of metastatic breast cancer with nab-paclitaxel in the community practice setting: a US oncology survey
Background Different dosages-schedules of nab-paclitaxel have been assessed in trials of metastatic breast cancer (MBC). However, there is limited information on nab-paclitaxel dosing-scheduling in the community setting.

 

Objective To report on experience with nab-paclitaxel for human epidermal growth factor receptor 2 (HER2)–negative MBC and identify patient characteristics affecting nab-paclitaxel treatment patterns in the community practice setting.

 

Methods From September 6-October 21, 2013, a 35-question, web-based survey on nab-paclitaxel dosing, toxicities leading to dose modifications, management, and treatment duration was sent to US Oncology network oncologists. Respondents were categorized by percentage of their patients with HER2-negative MBC who received nab-paclitaxel.

 

Results 104 of 428 oncologists responded; 84% were from large practices (≥16 oncologists), and 56% had a high level of experience using nab-paclitaxel. For first- and second-line treatment, 100 mg/m2 weekly was the most common starting dosage-schedule, followed by 125 mg/m2 weekly and 260 mg/m2 every 3 weeks (q3w); 150 mg/m2 weekly was used least frequently. Several factors, including select aggressive disease characteristics, were found to affect nab-paclitaxel dose selection. Weekly dosing was preferred in patients with select aggressive disease characteristics, whereas q3w dosing was commonly used in patients aged ≤50 years and those with good performance status. Differences in management styles among oncologists with high compared with infrequent nab-paclitaxel experience were also observed. Peripheral neuropathy and neutropenia were common dose-limiting toxicities.

 

Limitations Recall and response bias may be limitations of this study.

 

Conclusions In the community setting, nab-paclitaxel 100 mg/m2 weekly was the most commonly used starting dose for patients with HER2-negative MBC, including those with aggressive disease characteristics.

 

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The Journal of Community and Supportive Oncology - 13(5)
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173-180
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metastatic breast cancer, MBC, nab-paclitaxel, human epidermal growth factor receptor 2-negative, HER2–negative
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Background Different dosages-schedules of nab-paclitaxel have been assessed in trials of metastatic breast cancer (MBC). However, there is limited information on nab-paclitaxel dosing-scheduling in the community setting.

 

Objective To report on experience with nab-paclitaxel for human epidermal growth factor receptor 2 (HER2)–negative MBC and identify patient characteristics affecting nab-paclitaxel treatment patterns in the community practice setting.

 

Methods From September 6-October 21, 2013, a 35-question, web-based survey on nab-paclitaxel dosing, toxicities leading to dose modifications, management, and treatment duration was sent to US Oncology network oncologists. Respondents were categorized by percentage of their patients with HER2-negative MBC who received nab-paclitaxel.

 

Results 104 of 428 oncologists responded; 84% were from large practices (≥16 oncologists), and 56% had a high level of experience using nab-paclitaxel. For first- and second-line treatment, 100 mg/m2 weekly was the most common starting dosage-schedule, followed by 125 mg/m2 weekly and 260 mg/m2 every 3 weeks (q3w); 150 mg/m2 weekly was used least frequently. Several factors, including select aggressive disease characteristics, were found to affect nab-paclitaxel dose selection. Weekly dosing was preferred in patients with select aggressive disease characteristics, whereas q3w dosing was commonly used in patients aged ≤50 years and those with good performance status. Differences in management styles among oncologists with high compared with infrequent nab-paclitaxel experience were also observed. Peripheral neuropathy and neutropenia were common dose-limiting toxicities.

 

Limitations Recall and response bias may be limitations of this study.

 

Conclusions In the community setting, nab-paclitaxel 100 mg/m2 weekly was the most commonly used starting dose for patients with HER2-negative MBC, including those with aggressive disease characteristics.

 

Click on the PDF icon at the top of this introduction to read the full article.

 

Background Different dosages-schedules of nab-paclitaxel have been assessed in trials of metastatic breast cancer (MBC). However, there is limited information on nab-paclitaxel dosing-scheduling in the community setting.

 

Objective To report on experience with nab-paclitaxel for human epidermal growth factor receptor 2 (HER2)–negative MBC and identify patient characteristics affecting nab-paclitaxel treatment patterns in the community practice setting.

 

Methods From September 6-October 21, 2013, a 35-question, web-based survey on nab-paclitaxel dosing, toxicities leading to dose modifications, management, and treatment duration was sent to US Oncology network oncologists. Respondents were categorized by percentage of their patients with HER2-negative MBC who received nab-paclitaxel.

 

Results 104 of 428 oncologists responded; 84% were from large practices (≥16 oncologists), and 56% had a high level of experience using nab-paclitaxel. For first- and second-line treatment, 100 mg/m2 weekly was the most common starting dosage-schedule, followed by 125 mg/m2 weekly and 260 mg/m2 every 3 weeks (q3w); 150 mg/m2 weekly was used least frequently. Several factors, including select aggressive disease characteristics, were found to affect nab-paclitaxel dose selection. Weekly dosing was preferred in patients with select aggressive disease characteristics, whereas q3w dosing was commonly used in patients aged ≤50 years and those with good performance status. Differences in management styles among oncologists with high compared with infrequent nab-paclitaxel experience were also observed. Peripheral neuropathy and neutropenia were common dose-limiting toxicities.

 

Limitations Recall and response bias may be limitations of this study.

 

Conclusions In the community setting, nab-paclitaxel 100 mg/m2 weekly was the most commonly used starting dose for patients with HER2-negative MBC, including those with aggressive disease characteristics.

 

Click on the PDF icon at the top of this introduction to read the full article.

 

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The Journal of Community and Supportive Oncology - 13(5)
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Treatment of metastatic breast cancer with nab-paclitaxel in the community practice setting: a US oncology survey
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Treatment of metastatic breast cancer with nab-paclitaxel in the community practice setting: a US oncology survey
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metastatic breast cancer, MBC, nab-paclitaxel, human epidermal growth factor receptor 2-negative, HER2–negative
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Blinatumomab for hard-to-treat form of acute lymphoblastic leukemia

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Blinatumomab for hard-to-treat form of acute lymphoblastic leukemia

The US Food and Drug Administration (FDA) has granted accelerated approval to blinatumomab for the treatment of adult patients with relapsed/ refractory Philadelphia chromosome-negative precursor B-cell acute lymphoblastic leukemia (BCP-ALL).1 Blinatumomab is the first of a novel class of antibodies to receive regulatory approval; a bispecific antibody targeting both CD19, expressed on the surface of B cells, and CD3, on cytotoxic T cells. The approval was based on the findings of a single-arm, multicenter, open-label study in patients at high-risk of poor outcome, which showed a significant improvement of blinatumomab over other available therapies in this setting.2

 

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The Journal of Community and Supportive Oncology - 13(5)
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The US Food and Drug Administration (FDA) has granted accelerated approval to blinatumomab for the treatment of adult patients with relapsed/ refractory Philadelphia chromosome-negative precursor B-cell acute lymphoblastic leukemia (BCP-ALL).1 Blinatumomab is the first of a novel class of antibodies to receive regulatory approval; a bispecific antibody targeting both CD19, expressed on the surface of B cells, and CD3, on cytotoxic T cells. The approval was based on the findings of a single-arm, multicenter, open-label study in patients at high-risk of poor outcome, which showed a significant improvement of blinatumomab over other available therapies in this setting.2

 

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The US Food and Drug Administration (FDA) has granted accelerated approval to blinatumomab for the treatment of adult patients with relapsed/ refractory Philadelphia chromosome-negative precursor B-cell acute lymphoblastic leukemia (BCP-ALL).1 Blinatumomab is the first of a novel class of antibodies to receive regulatory approval; a bispecific antibody targeting both CD19, expressed on the surface of B cells, and CD3, on cytotoxic T cells. The approval was based on the findings of a single-arm, multicenter, open-label study in patients at high-risk of poor outcome, which showed a significant improvement of blinatumomab over other available therapies in this setting.2

 

Click on the PDF icon at the top of this introduction to read the full article.

 

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The Journal of Community and Supportive Oncology - 13(5)
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The Journal of Community and Supportive Oncology - 13(5)
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Blinatumomab for hard-to-treat form of acute lymphoblastic leukemia
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Blinatumomab for hard-to-treat form of acute lymphoblastic leukemia
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acute lymphoblastic leukemia, ALL, blinatumomab, Philadelphia chromosome-negative precursor, B-cell acute lymphoblastic leukemia, BCP-ALL, CD19, CD3, allo-hematopoietic stem-cell transplantation, allo-HSCT
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